COMPOUNDS / PSYCHEDELIC / DMT
PSYCHEDELIC

DMT

HIGH RISK
BEST FOR:Consciousness Exploration●●●●●●●●○○8/10
DMT · N,N-DIMETHYLTRYPTAMINE · N,N-DMT · DIMITRI · SPIRIT MOLECULE · DIMITRY
PsychedelicTryptamineSerotonergicEntheogenicShort-acting

A potent, short-acting psychedelic tryptamine producing intense visionary experiences, naturally found in plants and mammals.

ROUTE / DOSAGE
LOW30mg
STANDARD60-100mg
HIGH150mg+
Note: Requires MAOI co-administration (e.g., ayahuasca) for oral activity
CYCLE
Not suitable for frequent or daily use effects are intensely acute
BIOAVAILABILITY
negligible oral without MAOI moderate inhaled
ACTIVE DURATION
5-20 min inhaled 2-6h oral w/ MAOI (subjective)
STORAGE
Room temperature dry away from light protect from moisture and heat
HALF-LIFE
~12 minutes (plasma)
DURATION BREAKDOWN
Total5-20 min inhaled; 2-6h oral w/ MAOI
Onset20-40 sec inhaled; 30-60 min oral
Come up1-3 min inhaled; 30-60 min oral
Peak5-15 min inhaled; 1-2h oral
Offset2-5 min inhaled; 1-2h oral
After effects30-60 min inhaled; 2-4h oral
Duration varies by route, dose, and individual. Source: community reports.
§ 01 — EFFECTS
7 documented
Intense visual and auditory hallucinations
Rapid ego dissolution and entity encounters
Altered perception of time and space
Profound mystical or spiritual experiences
Short duration of action (5-20 minutes when smoked)
Increased heart rate and blood pressure
Enhanced emotional states and introspection
§ 02 — RISKS
Acute hypertension and tachycardia requiring cardiovascular monitoring in vulnerable populations
Psychological distress, panic, and acute anxiety during the experience
Potential triggering of psychotic episodes in individuals with predisposition to psychosis
Risk of serotonin syndrome when combined with serotonergic medications
Possible reactivation phenomena (flashbacks) after the acute experience
Harmful interactions with MAO inhibitors when used orally without proper dietary restrictions
§ 03 — INTERACTIONS
No interaction with your stack
VIEW STACK
CAUTION
Cannabis
Stimulants
AVOID · DANGEROUS
Lithium
Tramadol
§ 04 — SCIENCE

EEG-fMRI studies in healthy volunteers demonstrate that IV DMT (20 mg) produces robust increases in global functional connectivity, network disintegration, and compression of the principal cortical gradient, with effects correlating to 5-HT2A receptor density maps. Recent research has identified DMT levels in rodent brains comparable to classical neurotransmitters, challenging earlier assumptions of trace-only concentrations and suggesting a potential endogenous neurotransmitter role. Preclinical studies indicate DMT may protect against ischemia-reperfusion injury and neurodegeneration via sigma-1 receptor activation, and may promote neuroplasticity, neurogenesis, and immunomodulation. However, clinical evidence in humans remains limited, with most therapeutic data coming from observational studies or surrogate compounds like 5-MeO-DMT rather than controlled DMT trials. Acute sympathomimetic side effects, including hypertension and tachycardia, have been documented and may limit therapeutic applicability without coformulation strategies.

§ 06 — SOURCES
[1]
Human brain effects of DMT assessed via EEG-fMRI (PMID 36940333)
pubmed.ncbi.nlm.nih.gov ↗
[2]
Exploring DMT: Endogenous role and therapeutic potential (PMID 39832530)
pubmed.ncbi.nlm.nih.gov ↗
[3]
Significance of mammalian N,N-dimethyltryptamine (DMT): A 60-year-old debate (PMID 35695604)
pubmed.ncbi.nlm.nih.gov ↗
[4]
The protective effect of DMT against neurodegeneration (PMID 40541317)
pubmed.ncbi.nlm.nih.gov ↗
[5]
Reducing Cardiovascular Side Effects of DMT Using Beta-Blockers (PMID 40365386)
pubmed.ncbi.nlm.nih.gov ↗
[6]
PsychonautWiki - DMT
psychonautwiki.org ↗
Disclaimer. Protokol.wiki is an informational reference. Nothing on this site is medical advice. Compounds listed may be unapproved, unregulated, or illegal in your jurisdiction. Consult a licensed physician before use. Data compiled from peer-reviewed literature.
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