COMPOUNDS / METABOLIC / ACTOVEGIN
METABOLIC

ACTOVEGIN

MEDIUM RISK
BEST FOR:Recovery●●●●●○○○○○5/10
ACTOVEGIN · DEPROTEINIZED CALF BLOOD HEMODIALYSATE · DEPROTEINIZED HEMODERIVATIVE OF CALF BLOOD · CALF BLOOD EXTRACT
RecoveryAntioxidantNeuroprotectiveWound HealingBovine Extract

Deproteinized calf blood hemodialysate used for muscle injuries, diabetic neuropathy, and neuroprotection with uncertain efficacy.

ROUTE / DOSAGE
LOW200mg
STANDARD200-400mg
HIGH600mg
Note: 200mg tablets; typically 1-2 tablets 3x daily with meals
CYCLE
10-180 days depending on indication
BIOAVAILABILITY
~30-50% oral ~100% parenteral
ACTIVE DURATION
6-12h (subjective)
STORAGE
Room temperature (15-25°C) protect from light do not freeze injectable forms
HALF-LIFE
Not established (plasma)
DURATION BREAKDOWN
Total6-12h
Onset30min-2h
Come up1-3h
Peak2-6h
Offset4-8h
After effects12-24h
Duration varies by route, dose, and individual. Source: community reports.
§ 01 — EFFECTS
7 documented
Enhanced glucose uptake under ischemia
Improved mitochondrial respiration
Reduced IL-1beta and ROS production
Accelerated muscle injury recovery
Neuroprotective and anti-apoptotic effects
Improved wound healing
Antioxidant activity
§ 02 — RISKS
Anaphylactic shock reported in a cyclist (bovine origin hypersensitivity)
Higher incidence of recurrent ischemic stroke, TIA, or intracerebral hemorrhage in one stroke study
Allergic reactions due to bovine protein origin
Active compound(s) unidentified, limiting safety profiling
Uncertain clinical efficacy across multiple indications
§ 03 — INTERACTIONS
Interaction data not yet available for this compound.
§ 04 — SCIENCE

A 2022 systematic review of Actovegin in ischemic stroke (5 studies, 3879 patients) found no consistent evidence of improved survival, quality of life, or neurologic outcomes, and one study reported higher incidence of recurrent stroke or hemorrhage versus placebo. In sports medicine, a pilot study showed return to play 8 days earlier with Actovegin versus physiotherapy alone, and a 2020 review concluded clinical efficacy for muscle injuries was supported by limited evidence with a safe profile. In vitro studies demonstrate reduced inflammatory cytokine secretion and improved mitochondrial respiration in a diabetic mouse model. Actovegin showed efficacy and safety in randomized trials for diabetic peripheral neuropathy and is authorized for this indication in several countries. The active compound(s) have never been definitively identified, and pharmacokinetic studies are not feasible due to the physiological nature of its components.

§ 06 — SOURCES
[1]
Actovegin in the management of patients after ischemic stroke: A systematic review
pubmed.ncbi.nlm.nih.gov ↗
[2]
Our experience on Actovegin, is it cutting edge?
pubmed.ncbi.nlm.nih.gov ↗
[3]
Actovegin--Cutting-edge sports medicine or voodoo remedy?
pubmed.ncbi.nlm.nih.gov ↗
[4]
Update on the Role of Actovegin in Musculoskeletal Medicine: A Review of the Past 10 Years
pubmed.ncbi.nlm.nih.gov ↗
[5]
Pathogenetic treatments for diabetic peripheral neuropathy
pubmed.ncbi.nlm.nih.gov ↗
[6]
Pleiotropic neuroprotective and metabolic effects of Actovegin's mode of action
pubmed.ncbi.nlm.nih.gov ↗
[7]
Actovegin reduces PMA-induced inflammation on human cells
pubmed.ncbi.nlm.nih.gov ↗
[8]
Actovegin improves skeletal muscle mitochondrial respiration and functional aerobic capacity in a type 1 diabetic male murine model
pubmed.ncbi.nlm.nih.gov ↗
Disclaimer. Protokol.wiki is an informational reference. Nothing on this site is medical advice. Compounds listed may be unapproved, unregulated, or illegal in your jurisdiction. Consult a licensed physician before use. Data compiled from peer-reviewed literature.
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