COMPOUNDS / NOOTROPIC / SELANK
NOOTROPIC

SELANK

LOW RISK
BEST FOR:Anxiety●●●●●●●○○○7/10
SELANK · SELANK PEPTIDE · TUFTSIN ANALOG · THR-LYS-PRO-ARG-PRO-GLY-PRO · SELANKAMIDE · SEMAX ANALOG
NootropicAnxiolyticPeptideSoviet / RussianNeuropeptideTuftsin analog

Synthetic tuftsin analog peptide with anxiolytic and nootropic effects, developed in Russia and used clinically there.

ROUTE / DOSAGE
LOW300mcg
STANDARD600-900mcg
HIGH1500mcg+
Note: Standard clinical formulation is 0.15% nasal spray; 2-3 drops per nostril, 2-3x daily
CYCLE
10-14 day courses 1-3 weeks off between cycles
BIOAVAILABILITY
~90% intranasal (nose-to-brain delivery)
ACTIVE DURATION
4-6h (subjective)
STORAGE
Refrigerate at 2-8°C protect from light keep nasal spray sealed
HALF-LIFE
30-60 min (plasma)
DURATION BREAKDOWN
Total4-6h
Onset10-30min
Come up20-40min
Peak1-2h
Offset1-2h
After effects2-4h
Duration varies by route, dose, and individual. Source: community reports.
§ 01 — EFFECTS
7 documented
Anxiolytic (anxiety reduction without sedation)
Cognitive enhancement and memory support
Stress-protective effects
Reduction of pro-inflammatory cytokines
Neuroplasticity support via BDNF modulation
Attenuation of opioid withdrawal symptoms
Mood stabilization
§ 02 — RISKS
Limited human clinical trial data outside Russia; most evidence is preclinical
Potential GABAergic interactions with benzodiazepines or other CNS depressants
Unregulated sourcing may lead to contamination or dosing errors
Long-term safety data in humans is sparse
Intranasal administration may cause local irritation
§ 03 — INTERACTIONS
Interaction data not yet available for this compound.
§ 04 — SCIENCE

Selank has been studied primarily in Russian preclinical and limited clinical research. Human fMRI data (52 participants) show altered resting-state functional connectivity between the right amygdala and temporal cortex within 5-20 minutes of intranasal administration. Animal studies demonstrate anxiolytic effects comparable to diazepam, attenuation of morphine withdrawal signs, protection against ethanol-induced memory impairment via BDNF regulation, and reduction of stress-induced pro-inflammatory cytokines. Gene expression studies confirm modulation of GABAergic neurotransmission genes. However, large-scale randomized clinical trials outside Russia are lacking, and most evidence comes from small animal models and in vitro studies. The peptide is approved for clinical use in Russia but lacks regulatory approval in most other countries.

§ 06 — SOURCES
[1]
Functional Connectomic Approach to Studying Selank and Semax Effects
pubmed.ncbi.nlm.nih.gov ↗
[2]
Therapeutic Peptides in Orthopaedics: Applications, Challenges, and Future Directions
pubmed.ncbi.nlm.nih.gov ↗
[3]
Selank, a Peptide Analog of Tuftsin, Attenuates Aversive Signs of Morphine Withdrawal in Rats
pubmed.ncbi.nlm.nih.gov ↗
[4]
Effect of Selank on Morphological Parameters of Rat Liver in Chronic Foot-Shock Stress
pubmed.ncbi.nlm.nih.gov ↗
[5]
The Influence of Selank on the Level of Cytokines Under the Conditions of Social Stress
pubmed.ncbi.nlm.nih.gov ↗
[6]
Selank, Peptide Analogue of Tuftsin, Protects Against Ethanol-Induced Memory Impairment by Regulating BDNF Content
pubmed.ncbi.nlm.nih.gov ↗
[7]
Selank Administration Affects the Expression of Some Genes Involved in GABAergic Neurotransmission
pubmed.ncbi.nlm.nih.gov ↗
[8]
GABA, Selank, and Olanzapine Affect the Expression of Genes Involved in GABAergic Neurotransmission in IMR-32 Cells
pubmed.ncbi.nlm.nih.gov ↗
Disclaimer. Protokol.wiki is an informational reference. Nothing on this site is medical advice. Compounds listed may be unapproved, unregulated, or illegal in your jurisdiction. Consult a licensed physician before use. Data compiled from peer-reviewed literature.
METHODOLOGYCONTRIBUTECONTACT