COMPOUNDS / SARM / LGD-4033
SARM

LGD-4033

HIGH RISK
BEST FOR:Muscle Growth●●●●●●●●○○8/10
LGD-4033 · LIGANDROL · VK5211 · ANABOLICUM
SARMAndrogenicMuscle GrowthWADA BannedResearch Chemical

A selective androgen receptor modulator developed for muscle-wasting conditions, widely used off-label for muscle growth despite documented hepatotoxicity and endocrine suppression.

ROUTE / DOSAGE
LOW1mg
STANDARD5-10mg
HIGH20mg+
Note: Once daily; clinical trials used 1-22mg. Higher doses increase risk of testosterone suppression and hepatotoxicity
CYCLE
6-8 weeks on 4-8 weeks off
BIOAVAILABILITY
high oral (~100%)
ACTIVE DURATION
24-36h (subjective)
STORAGE
Room temperature dry away from light
DURATION BREAKDOWN
Total24-36h
Onset1-3h
Come up1-2h
Peak2-6h
Offset12-24h
After effects24-48h
Duration varies by route, dose, and individual. Source: community reports.
§ 01 — EFFECTS
7 documented
Increased lean body mass
Enhanced muscular strength
Increased body weight
Suppressed testosterone production
Altered lipid profiles
Elevated liver enzymes
Improved muscle recovery
§ 02 — RISKS
Drug-induced liver injury including cholestatic hepatitis and fibrosis
Severe suppression of total and free testosterone with reduced FSH and SHBG
Decreased bone mineral density and bone mineral content
Adverse lipid changes: increased LDL and triglycerides, decreased HDL
Elevated AST and ALT liver enzymes up to 2-3x baseline
Psychiatric symptoms reported with off-label SARM use
§ 03 — INTERACTIONS
Interaction data not yet available for this compound.
§ 04 — SCIENCE

A Phase I clinical trial demonstrated dose-dependent increases in lean body mass with LGD-4033 at doses of 0.1-1mg over 21 days, with a half-life of approximately 24-36 hours. A case report of 10mg daily for 5 weeks showed increased body mass and lean mass but also significant suppression of total and free testosterone, elevated liver enzymes, worsened lipid profiles, and decreased bone mineral density. Multiple case reports document drug-induced liver injury (DILI) including cholestatic hepatitis with fibrosis. Anti-doping studies have identified numerous metabolites detectable in urine for up to 21 days post-administration. Evidence is limited to small clinical trials, case reports, and anti-doping metabolism studies; no large-scale human safety trials exist.

§ 06 — SOURCES
[1]
LGD-4033 and MK-677 use impacts body composition, circulating biomarkers, and skeletal muscle androgenic hormone and receptor content: A case report
pubmed.ncbi.nlm.nih.gov ↗
[2]
Ligandrol (LGD-4033)-Induced Liver Injury
pubmed.ncbi.nlm.nih.gov ↗
[3]
LGD-4033 and a Case of Drug-Induced Liver Injury: Exploring the Clinical Implications of Off-Label Selective Androgen Receptor Modulator Use in Healthy Adults
pubmed.ncbi.nlm.nih.gov ↗
[4]
Variation of Sequential Ligandrol (LGD-4033) Metabolite Levels in Routine Anti-Doping Urine Samples Detected with or without Other Xenobiotics
pubmed.ncbi.nlm.nih.gov ↗
[5]
Human in vivo metabolism study of LGD-4033 (Erratum)
pubmed.ncbi.nlm.nih.gov ↗
[6]
Human in vivo metabolism study of LGD-4033
pubmed.ncbi.nlm.nih.gov ↗
[7]
Investigations into the elimination profiles and metabolite ratios of micro-dosed selective androgen receptor modulator LGD-4033 for doping control purposes
pubmed.ncbi.nlm.nih.gov ↗
[8]
Investigations into the concentration and metabolite profiles of stanozolol and LGD-4033 in blood plasma and seminal fluid using liquid chromatography high-resolution mass spectrometry
pubmed.ncbi.nlm.nih.gov ↗
Disclaimer. Protokol.wiki is an informational reference. Nothing on this site is medical advice. Compounds listed may be unapproved, unregulated, or illegal in your jurisdiction. Consult a licensed physician before use. Data compiled from peer-reviewed literature.
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