COMPOUNDS / PSYCHEDELIC / AL-LAD
PSYCHEDELIC

AL-LAD

MEDIUM RISK
BEST FOR:Exploration●●●●●○○○○○5/10
AL-LAD · N6-ALLYL-6-NORLYSERGIC ACID DIETHYLAMIDE · 6-ALLYL-NOR-LSD · N-ALLYL-NOR-LSD · 6-ALLYL-NOR-LSD
PsychedelicLysergamideLSD analogResearch Chemical5-HT2A

N6-allyl LSD analog and lysergamide psychedelic acting primarily at 5-HT2A receptors, slightly less potent than LSD.

ROUTE / DOSAGE
LOW50-100mcg
STANDARD100-225mcg
HIGH225-350mcg
Note: Typically administered via blotter paper; swallow after brief sublingual hold
CYCLE
Max 1x per week 2+ week tolerance reset
BIOAVAILABILITY
high oral ~100% sublingual
ACTIVE DURATION
6-10h (subjective)
STORAGE
Cool dry away from light blotter degrades with heat and UV exposure
HALF-LIFE
3-5h (plasma)
DURATION BREAKDOWN
Total6-10 hours
Onset20-60 min
Come up30-60 min
Peak2-4 hours
Offset2-3 hours
After effects2-6 hours
Duration varies by route, dose, and individual. Source: community reports.
§ 01 — EFFECTS
7 documented
Visual distortions and hallucinations
Enhanced color perception
Altered sense of time and space
Euphoria and mood elevation
Introspective and philosophical thinking
Auditory enhancements
Mild stimulant-like energy
§ 02 — RISKS
Fatal ventricular dysrhythmia reported in association with AL-LAD use
Psychological distress including anxiety, paranoia, and panic reactions
Risk of triggering latent psychiatric conditions such as psychosis
Significant cross-tolerance with LSD and other 5-HT2A psychedelics
Potential for difficult or overwhelming experiences at high doses
Unknown long-term safety profile due to absence of clinical trials
§ 03 — INTERACTIONS
No interaction with your stack
VIEW STACK
CAUTION
Cannabis
Stimulants
AVOID · DANGEROUS
Lithium
Tramadol
§ 04 — SCIENCE

AL-LAD emerged on the new psychoactive substances market in 2013 and has been analytically characterized using NMR, GC-MS, and LC-MS methods. Behavioral studies in mice confirmed 5-HT2A-mediated head-twitch responses with potency slightly below LSD. In vitro metabolism studies of prodrug derivatives (1P-AL-LAD, 1cP-AL-LAD) confirmed conversion to AL-LAD as the primary metabolite. A published case report documented fatal ventricular dysrhythmia associated with AL-LAD use, though causality in isolation remains uncertain. Human pharmacokinetic data are extremely limited, and most potency comparisons are derived from rodent models, limiting direct translation to human dosing. No controlled clinical trials have been conducted.

§ 06 — SOURCES
[1]
Analytical profile of the lysergamide 1cP-AL-LAD and detection of impurities
pubmed.ncbi.nlm.nih.gov ↗
[2]
Analytical profile, in vitro metabolism and behavioral properties of the lysergamide 1P-AL-LAD
pubmed.ncbi.nlm.nih.gov ↗
[3]
Synthesis and analytical characterization of 1T-AL-LAD
pubmed.ncbi.nlm.nih.gov ↗
[4]
Spatiotemporal Mapping of Online Interest in Cannabis and Popular Psychedelics in Poland
pubmed.ncbi.nlm.nih.gov ↗
[5]
Identification of 1S-LSD and 1T-AL-LAD in paper sheet products
pubmed.ncbi.nlm.nih.gov ↗
[6]
Return of the lysergamides Part II: Analytical and behavioural characterization of AL-LAD and LSZ
pubmed.ncbi.nlm.nih.gov ↗
[7]
Case of fatal ventricular dysrhythmia associated with AL-LAD
pubmed.ncbi.nlm.nih.gov ↗
[8]
Return of the lysergamides Part III: Analytical characterization of ETH-LAD and 1P-ETH-LAD
pubmed.ncbi.nlm.nih.gov ↗
[9]
PsychonautWiki - AL-LAD
psychonautwiki.org ↗
Disclaimer. Protokol.wiki is an informational reference. Nothing on this site is medical advice. Compounds listed may be unapproved, unregulated, or illegal in your jurisdiction. Consult a licensed physician before use. Data compiled from peer-reviewed literature.
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